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Status |
Public on Jun 08, 2015 |
Title |
Synergism between PPARĪ± and glucocorticoid receptor signaling promotes self-renewal of BFU-E erythroid progenitors and increases red cell production [ChIP-seq] |
Organism |
Mus musculus |
Experiment type |
Genome binding/occupancy profiling by high throughput sequencing
|
Summary |
ChIP-Seq analyses of GR and PPARa occupancy in mouse E14.5 fetal liver BFU-E cells untreated or treated by DEX with or without GW7647
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Overall design |
ChIP-Seq on GR and PPAR alpha in purified 10^7 mouse BFU-E cells purified from E14.5 fetal livers with or without treatment of Dexamethasone and/or GW7647
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Contributor(s) |
Lee H, Gao X, Barrasa MI, Li H, Elmes RR, Peters LL, Lodish HF |
Citation(s) |
25970251 |
Submission date |
Oct 28, 2014 |
Last update date |
May 15, 2019 |
Contact name |
Xiaofei Gao |
E-mail(s) |
xgao@wi.mit.edu, wibr-bioinformatics@wi.mit.edu
|
Organization name |
Whitehead Institute for Biomedical Research
|
Lab |
Lodish
|
Street address |
9CC
|
City |
Cambridge |
State/province |
MA |
ZIP/Postal code |
02142 |
Country |
USA |
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Platforms (1) |
GPL17021 |
Illumina HiSeq 2500 (Mus musculus) |
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Samples (7)
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GSM1533397 |
Glucocorticoid receptor_ChIP-seq_untreated |
GSM1533398 |
PPAR alpha_ChIP-seq_untreated |
GSM1533399 |
Glucocorticoid receptor_ChIP-seq_Dexamethasone |
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This SubSeries is part of SuperSeries: |
GSE63837 |
Synergism between PPARĪ± and glucocorticoid receptor signaling promotes self-renewal of BFU-E erythroid progenitors and increases red cell production |
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Relations |
BioProject |
PRJNA265113 |
SRA |
SRP049353 |