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Items: 8

1.

The effect of TAZ-K in neonatal rat cardiomyocytes

(Submitter supplied) Accumulating evidence suggests that new cardiomyocytes are generated from existing cardiomyocytes at a low rate throughout life. This limited regenerative capacity is unable to restore cardiac function after substantial cardiac damage; thus, small molecules have been examined for their ability to enhance the replication of existing cardiomyocytes. A novel compound TAZ-K promoted cardiomyocyte proliferation, therefore, we analyzed the effect of TAZ-K on gene expression in neonatal rat cardiomyocytes. more...
Organism:
Rattus norvegicus
Type:
Expression profiling by high throughput sequencing
Platform:
GPL18694
6 Samples
Download data: TXT
Series
Accession:
GSE95274
ID:
200095274
2.

Illumina HiSeq 2500 (Rattus norvegicus)

Organism:
Rattus norvegicus
484 Series
13096 Samples
Download data
Platform
Accession:
GPL18694
ID:
100018694
3.

TAZ-K treated cardiomyocytes rep3

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501146
ID:
302501146
4.

TAZ-K treated cardiomyocytes rep2

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501145
ID:
302501145
5.

TAZ-K treated cardiomyocytes rep1

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501144
ID:
302501144
6.

non-treated cardiomyocytes rep3

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501143
ID:
302501143
7.

non-treated cardiomyocytes rep2

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501142
ID:
302501142
8.

non-treated cardiomyocytes rep1

Organism:
Rattus norvegicus
Source name:
cardiomyocytes
Platform:
GPL18694
Series:
GSE95274
Download data
Sample
Accession:
GSM2501141
ID:
302501141
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Supplemental Content

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