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PubMed (cited) for id: 606806
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Author Correction: Histidine catabolism is a major determinant of methotrexate sensitivity.
Nature. 2022 Feb;602(7895):E17-E18. doi: 10.1038/s41586-021-03487-2.
Nature. 2022.
PMID: 35017686
No abstract available.
Allelic spectrum of formiminotransferase-cyclodeaminase gene variants in individuals with formiminoglutamic aciduria.
Majumdar R, Yori A, Rush PW, Raymond K, Gavrilov D, Tortorelli S, Matern D, Rinaldo P, Feldman GL, Oglesbee D.
Majumdar R, et al.
Mol Genet Genomic Med. 2017 Nov;5(6):795-799. doi: 10.1002/mgg3.333. Epub 2017 Sep 11.
Mol Genet Genomic Med. 2017.
PMID: 29178637
Free PMC article.
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The molecular basis of glutamate formiminotransferase deficiency.
Hilton JF, Christensen KE, Watkins D, Raby BA, Renaud Y, de la Luna S, Estivill X, MacKenzie RE, Hudson TJ, Rosenblatt DS.
Hilton JF, et al.
Hum Mutat. 2003 Jul;22(1):67-73. doi: 10.1002/humu.10236.
Hum Mutat. 2003.
PMID: 12815595
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Cloning and characterization of human FTCD on 21q22.3, a candidate gene for glutamate formiminotransferase deficiency.
Solans A, Estivill X, de la Luna S.
Solans A, et al.
Cytogenet Cell Genet. 2000;88(1-2):43-9. doi: 10.1159/000015483.
Cytogenet Cell Genet. 2000.
PMID: 10773664
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Formiminotransferase cyclodeaminase is an organ-specific autoantigen recognized by sera of patients with autoimmune hepatitis.
Lapierre P, Hajoui O, Homberg JC, Alvarez F.
Lapierre P, et al.
Gastroenterology. 1999 Mar;116(3):643-9. doi: 10.1016/s0016-5085(99)70186-1.
Gastroenterology. 1999.
PMID: 10029623
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