Targeted gene disruption in endocrine research--the case of glucagon-like peptide-1 and neuroendocrine function

Endocrinology. 2000 Feb;141(2):473-5. doi: 10.1210/endo.141.2.7372.
No abstract available

Publication types

  • Comment
  • Editorial
  • Review

MeSH terms

  • Animals
  • Gene Deletion*
  • Glucagon / deficiency
  • Glucagon / genetics
  • Glucagon / physiology*
  • Glucagon-Like Peptide 1
  • Glucagon-Like Peptide-1 Receptor
  • Mice
  • Mice, Knockout
  • Neurosecretory Systems / physiology*
  • Peptide Fragments / deficiency
  • Peptide Fragments / genetics
  • Peptide Fragments / physiology*
  • Protein Precursors / deficiency
  • Protein Precursors / genetics
  • Protein Precursors / physiology*
  • Receptors, Glucagon / deficiency
  • Receptors, Glucagon / genetics
  • Receptors, Glucagon / physiology

Substances

  • Glp1r protein, mouse
  • Glucagon-Like Peptide-1 Receptor
  • Peptide Fragments
  • Protein Precursors
  • Receptors, Glucagon
  • Glucagon-Like Peptide 1
  • Glucagon