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Links from GEO DataSets

Items: 4

1.
Full record GDS4833

Cediranib effect on metastatic alveolar soft part sarcoma

Analysis of metastatic alveolar soft part sarcoma (ASPS) biopsies from patients treated with cediranib, a small-molecule inhibitor of vascular endothelial growth factor receptors. Results provide insight into molecular mechanisms underlying the substantial single-agent activity of cediranib on ASPS.
Organism:
Homo sapiens
Type:
Expression profiling by array, transformed count, 2 agent, 6 individual sets
Platform:
GPL570
Series:
GSE32569
12 Samples
Download data: CEL
2.

Expression data for Cediranib in Metastatic ASPS

(Submitter supplied) Gene expression from pre- and post- Cediranib treated patients with metastatic Alveolar Soft Part Sarcoma (ASPS)
Organism:
Homo sapiens
Type:
Expression profiling by array
Dataset:
GDS4833
Platform:
GPL570
12 Samples
Download data: CEL
Series
Accession:
GSE32569
ID:
200032569
3.

Gene Expression Profiling of Alveolar Soft-Part Sarcoma (ASPS)

(Submitter supplied) Alveolar soft-part sarcoma (ASPS) is an extremely rare, highly vascular soft tissue sarcoma affecting predominantly adolescents and young adults. In an attempt to gain insight into the pathobiology of this enigmatic tumor, we performed the first genome-wide gene expression profiling study.
Organism:
Homo sapiens
Type:
Expression profiling by array
Platform:
GPL570
16 Samples
Download data: CEL, CHP
Series
Accession:
GSE13433
ID:
200013433
4.

Malignant melanotic Xp11 neoplasms exhibit a clinicopathological spectrum and gene expression profiling akin to alveolar soft part sarcoma: a proposal for reclassification

(Submitter supplied) We employed RNA sequencing-based clustering analysis to better understand the relationship of this unique entity to its closest histopathologic and immunophenotypic counterparts, including ASPS, conventional PEComa, and Xp11 translocation RCC
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platform:
GPL20301
51 Samples
Download data: TXT
Series
Accession:
GSE150474
ID:
200150474
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Supplemental Content

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